mouse il-5 elisa kit (Multi Sciences (Lianke) Biotech Co Ltd)
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Multi Sciences (Lianke) Biotech Co Ltd
mouse il-5 elisa kit
Mouse Il 5 Elisa Kit, supplied by Multi Sciences (Lianke) Biotech Co Ltd, used in various techniques. Bioz Stars score: 94/100, based on 90 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/il+5/Mouse+IL-5+ELISA+Kit/custom%40ek205%4042107577
Average 94 stars, based on 90 article reviews
Mouse Il 5 Elisa Kit, supplied by Multi Sciences (Lianke) Biotech Co Ltd, used in various techniques. Bioz Stars score: 94/100, based on 90 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/il+5/Mouse+IL-5+ELISA+Kit/custom%40ek205%4042107577
Average 94 stars, based on 90 article reviews
mouse il-5 elisa kit - by Bioz Stars,
2026-09
94/100 stars
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Enzyme-linked Immunosorbent Assay:Article Title: Increased SOAT2 expression in aged regulatory T cells is associated with altered cholesterol metabolism and reduced anti-tumor immunity. Article Snippet: After restimulation with PMA (#HY-18739, MCE) + Ionomycin (#HY13434, MCE) for 6 h, cytokine levels as following: IL-2 (#EK102HS, #EK202HS, MULTI SCIENCES), IL-10 (#EK110, #EK210, MULTI SCIENCES), IL-35 (#E-EL-H2443c, #E-EL-M0733c, Elabscience) and TGF-β (#EK981, MULTI SCIENCES) in Treg cells, was assessed by ELISA kits in the supernatants of the culture medium according to the product instruction. .. As well as Cytokine with ELISA kits was performed for assessment of IL-1β (#EK101B, EK201B, MULTI SCIENCES), IL-2 (#EK102HS, #EK202HS, MULTI SCIENCES), IL-4 (#EK104, #EK204, MULTI SCIENCES), Article Title: NEDD4 like E3 ubiquitin protein ligase-mediated ubiquitination and degradation of dipeptidyl peptidase 4 restrains Th2-linked inflammation in allergic rhinitis. Article Snippet: Allergic rhinitis (AR) is a chronic inflammatory disorder characterized by type 2 T helper (Th2) cell-dominant immune responses.. NEDD4 like E3 ubiquitin protein ligase (NEDD4L) plays a role in regulating T cell activation and Th cell differentiation.. Dipeptidyl peptidase 4 (DPP4) is involved in Th2 inflammatory responses in AR. Article Title: Increased SOAT2 expression in aged regulatory T cells is associated with altered cholesterol metabolism and reduced anti-tumor immunity Article Snippet: After restimulation with PMA (#HY-18739, MCE) + Ionomycin (#HY-13434, MCE) for 6 h, cytokine levels as following: IL-2 (#EK102HS, #EK202HS, MULTI SCIENCES), IL-10 (#EK110, #EK210, MULTI SCIENCES), IL-35 (#E-EL-H2443c, #E-EL-M0733c, Elabscience) and TGF-β (#EK981, MULTI SCIENCES) in Treg cells, was assessed by ELISA kits in the supernatants of the culture medium according to the product instruction. .. As well as Cytokine with ELISA kits was performed for assessment of IL-1β (#EK101B, EK201B, MULTI SCIENCES), IL-2 (#EK102HS, #EK202HS, MULTI SCIENCES), IL-4 (#EK104, #EK204, MULTI SCIENCES), Article Title: Mechanism of action of M-XQLD treatment for asthma: role of STARD13 in Th17 suppression. Article Snippet: asthma and cause airway basement membrane thickening, increased deposition of collagen, and fibrosis [2].. Therapeutic drugs for asthma include bronchodilators, anti-allergic drugs, and corticosteroids.. However, the prognosis of asthma still poor, and present potentially serious side effect [3]. Article Title: DUSP14 attenuates airway inflammation and mucus hypersecretion in allergic asthma by regulating TAK1 activity. Article Snippet: Allergic asthma is characterized by persistent chronic airway inflammation, leading to mucus hypersecretion and airway hyperresponsiveness.. Dual-specificity phosphatase 14 (DUSP14), a member of the DUSP family, is a key regulator in various biological processes.. However, the function of DUSP14 in allergic asthma remains to be elucidated. Article Title: Microplastic exposure induces HSP90α secretion and aggravates asthmatic airway remodeling via PI3K-Akt-mTOR pathway. Article Snippet: .. The levels of IL-4 (EK204, Multi Sciences, China), Article Title: Asthmatic Pulmonary Fibroblasts Aggravate Airway Inflammation Through FKBP5-Dependent Necroptosis Article Snippet: Airway inflammation is a hallmark pathological feature of bronchial asthma and is closely associated with airway hyperresponsiveness.. Pulmonary fibroblasts are key contributors to asthmatic inflammation, yet the underlying mechanisms remain insufficiently defined.. In this study, FK506 binding protein 51 (FKBP5) was significantly upregulated in fibroblasts from individuals with asthma, and its expression was positively correlated with disease severity. |
